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K562

K562

K562

K562

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Conclusion: The growth inhibition and apoptosis induction of K562 cells treated by DFMO were associated with suppressed telomerase activity .

结论 DFMO 引起的 K562 细胞生长抑制及凋亡诱导与端粒酶活性抑制有关。

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Conclusion Etoposide chemotherapy combined with hyperthermia showed obvious inhibitive effect to K562 cells .

结论热疗联合足叶乙甙能增强对 K562 细胞的体外抑制作用；

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The K562 cells treated with toosendanin extracts showed morphological characteristics of apoptotic cells .

处理组细胞可见典型的凋亡形态学改变；

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Result : Tricholoma Matsutake markedly inhibited K562 cell proliferation .

结果：松茸对 K562 细胞增殖具有明显的抑制作用。

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Objective To observe the effects of Triptolide on proliferation and apoptosis of erythroleukemia K562 in vitro.

目的探讨雷公藤内酯醇对人类红白血病 K562 细胞株增殖和凋亡的影响。

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K562 cells under iron deprivation was also confirmed by western blot analysis .

K562 细胞剥夺下铁也证实了免疫印迹分析。

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MTT method to observe the effects of different concentrations of serum and Rubus parvifolius saponin on K562 cell proliferation .

MTT 法观察不同浓度的含药血清和茅莓总皂苷对 K562 细胞株的增殖作用的影响。

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